
White Phosphorus (WP), known as Willy Pete, is used for signaling, screening, and incendiary purposes. White Phosphorus can be used to destroy the enemy's equipment or to limit his vision. It is used against vehicles, petroleum, oils and lubricants (POL) and ammunition storage areas, and enemy observers.
(copied from http://www.globalsecurity.org/military/systems/munitions/wp.htm)
However as you can see below pictures, the White Phosphorus were used to hit children. The pictures below are the attack on UNRWA SCHOOL in Beit Lahia, Gaza, PALESTINE. Make sure you read till last to know what is white phosphorus! and the effect!






Throw removed phosphorus or clothing affected by phosphorus into water or allow to bum in suitable location. Cover phosphorus burns with moist dressing and keep moist to prevent renewed inflammation. It is neccessary to dress white phosphorus-injured patients with saline-soaked dressings to prevent reignition of the phosphorus by contact with the air.
Some nations recommend washing the skin with a 0.5-2.0% copper sulphate solution or a copper sulphate impregnated pad. Wounds may be rinsed with a 0.1%-0.2% copper sulphate solution, if available. Dark coloured deposits may be removed with forceps. Prevent prolonged contact of any copper sulphate preparations with the tissues by prompt, copious flushing with water or saline, as there is a definite danger of copper poisoning. It may be necessary to repeat the first aid measures to completely remove all phosphorus.






White Phosphorus (WP) - Other Health Effects
Systemic toxicity may occur if therapy is not administered. Therapy consists of topical use of a bicarbonate solution to neutralize phosphoric acids and mechanical removal and debridement of particles. A Wood’s lamp in a darkened room may help to identify remaining luminescent particles. The early signs of systemic intoxication by phosphorus are abdominal pain, jaundice, and a garlic odor of the breath; prolonged intake may cause anemia, as well as cachexia and necrosis of bone, involving typically the maxilla and mandible (phossy jaw). Prolonged absorption of phosphorus causes necrosis of bones. It is a hepatotoxin.
The presenting complaints of overexposed workers may be toothache and excessive salivation. There may be a dull red appearance of the oral mucosa. One or more teeth may loosen, with subsequent pain and swelling of the jaw; healing may be delayed following dental procedures such as extractions; with necrosis of bone, a sequestrum may develop with sinus tract formation. In a series of 10 cases, the shortest period of exposure to phosphorus fume (concentrations not measured) that led to bone necrosis was 10 months (two cases), and the longest period of exposure was 18 years.
Signs and symptoms include irritation of the eyes and the respiratory tract; abdominal pain, nausea, and jaundice; anemia, cachexia, pain, and loosening of teeth, excessive salivation, and pain and swelling of the jaw; skin and eye burns. Phossy jaw must be differentiated from other forms of osteomyelitis. With phossy jaw, a sequestrum forms in the bone and is released from weeks to months later; the sequestra are light in weight, yellow to brown, osteoporotic, and decalcified, whereas sequestra from acute staphylococcal osteomyelitis are sharp, white spicules of bone, dense and well calcified. In acute staphylococcal osteomyelitis, the radiographic picture changes rapidly and closely follows the clinical course, but with phossy jaw the diagnosis sometimes is clinically obvious before radiological changes are discernible. It is good dental practice to take routine X-ray films of jaws, but experience indicates that necrosis can occur in the absence of any pathology that is visible on the roentgenogram.
More Info: http://www.atsdr.cdc.gov/tfacts103.pdf
1 comments:
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